Analytical Data
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基因名
MRPL34
- Application
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别名
MRPL34; 39S ribosomal protein L34; mitochondrial; L34mt; MRP-L34; Mitochondrial large ribosomal subunit protein bL34m
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种属
Human
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表达系统
E. coli
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标签
GST-tag at N-terminal
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纯度
Greater than 90% as determined by SDS-PAGE.
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蛋白编号
Q9BQ48
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表达区间
1-92 aa
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氨基酸序列
MAVLAGSLLGPTSRSAALLGGRWLQPRAWLGFPDAWGLPTPQQARGKARGNEYQPSNIKRKNKHGWVRRLSTPAGVQVILRRMLKGRKSLSH
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分子量
36.6 kDa
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内毒素
< 1.0 EU per μg protein as determined by the LAL method.
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性状
Freeze-dried powder
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缓冲液
PBS, pH7.4, containing 0.01% SKL, 1mM DTT, 5% Trehalose and Proclin300.
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复溶方法
Reconstitute in ddH2O to a concentration of 0.1-0.5 mg/mL. Do not vortex.
- 个性化定制
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稳定性测试
The thermal stability is described by the loss rate. The loss rate was determined by accelerated thermal degradation test, that is, incubate the protein at 37℃ for 48h, and no obvious degradation and precipitation were observed. The loss rate isless than 8% within the expiration date under appropriate storage condition.
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保存条件 & 期限
Samples are stable for up to twelve months from date of receipt at -20℃ to -80℃. Store it under sterile conditions at -20℃ to -80℃. It is recommended that the protein be aliquoted for optimal storage. Avoid repeated freeze-thaw cycles.
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运输条件
In general, recombinant proteins are supplied as lyophilized powder and shipped at ambient temperature. For bulk packages, the proteins are provided as frozen liquid and shipped with blue ice, unless otherwise requested by the customer.
Quality inspection process
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Protein Description
MRPL34, a gene encoding a mitochondrial ribosomal protein, plays a significant role in the synthesis of mitochondrial proteins, which are essential for cellular respiration and energy production. Research into MRPL34 is particularly relevant due to its involvement in mitochondrial function and its potential implications in various diseases, including mitochondrial myopathies and metabolic disorders. As mitochondrial dysfunction is increasingly recognized as a contributing factor in a range of pathologies, understanding the role of MRPL34 can provide insights into the molecular mechanisms driving these conditions. Recent studies have suggested that mutations or alterations in MRPL34 can disrupt mitochondrial translation and lead to impaired energy metabolism, further highlighting its importance in maintaining cellular homeostasis. Additionally, exploring the structure and function of MRPL34 may aid in identifying therapeutic targets for conditions associated with mitochondrial dysfunction. Therefore, MRPL34 serves as a critical focus in mitochondrial biology, with implications for both basic research and clinical applications aimed at addressing mitochondrial-related diseases.
